![]() ![]() ![]() Surgical intervention or renal denervation may be considered in resistant cases.Ī 21 year old woman with no prior medical illness presented with epistaxis and raised blood pressure of 200/142 mmHg. Hormonal evaluation should be undertaken to determine whether its presence contributes to hypertension in the patient as targeted treatment such as aldosterone antagonist can be initiated. ![]() ConclusionĪccessory renal arteries are a potential cause renovascular hypertension which can be detected via CT angiography or magnetic resonance angiography. Imaging studies showed the presence of accessory renal artery. Both patients presented with hypokalemia and further investigations revealed hyperaldosteronism with unsuppressed renin levels. We report 2 cases of hypertension with secondary hyperaldosteronism associated with accessory renal arteries. Accessory renal arteries are variants in the vascular anatomy which are often thought to be innocuous but in some circumstances can cause renovascular hypertension leading to secondary hyperaldosteronism. Hypokalemia in the presence of hypertension is often attributed to primary hyperaldosteronism as a cause of secondary hypertension, however secondary hyperaldosteronism may present similarly.
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